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The author:(作者)delv
published in(发表于) 2012/5/11 1:45:57
The AIDS virus spread in the body is an accomplice

Xinhua net Madrid April 26 (reporter XieYuZhi) Spanish scientists involved in a new study found that a molecular indulge in the AIDS virus in the human body the process of transmission ACTS as a "accomplice". The discovery could help to prevent the spread of the AIDS virus developed drugs.
Spain advanced chemical institute and Germany Catalan institutions such as the university researchers found that AIDS virus shell surface ganglion (molecular is to enable the virus were invading the body in the body, and then the dendritic cells spread "accomplice". Further experiments, structure similar but not including the molecules of man-made virus samples particles can't enter the dendritic cells. They think accordingly, remove ganglion glucoside ester will effectively restrain the AIDS virus spread.
The researchers introduction, dendritic cells in the human body each fill in the organization "patrol", capture pathogens "intruder" and the crushing, "sample" into lymphoid tissue, initiate relevant immune response. But for the AIDS virus, because the existence of ganglion glucoside ester, dendritic cells cannot be divided, so the virus to complete invading the immune system, which is equal to the immune system to set a allows the AIDS virus of free access "Trojan horse". Once in lymphatic, the AIDS virus will kill the body's immune cells.
Researchers in the new issue network science magazine "science public library", the future is expected to development organization according to the spread of HIV/AIDS new drug, with commonly used "cocktails" to combat AIDS virus. But due to the need for the full clinical trials, the development of new drugs on the market and at least still need more than ten years.
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新华网马德里４月２６日电（报道员谢宇智）西班牙科学家参与的一项新研究发现，一种分子在纵容艾滋病病毒在人体内传播过程中充当了“帮凶”。这一发现有助于开发阻止艾滋病病毒扩散的药物。
西班牙加泰罗尼亚高级化学研究所和德国海德堡大学等机构的研究人员发现，艾滋病病毒外壳表面的神经节苷酯分子是使病毒侵入人体树突状细胞、进而在体内传播的“帮凶”。进一步实验证实，构造相似但不含这种分子的人造病毒样颗粒无法进入树突状细胞。他们据此认为，去除神经节苷酯将有效抑制艾滋病病毒扩散。
研究人员介绍，树突状细胞平日在人体各组织内“巡逻”，捕捉病原体“入侵者”并将其粉碎，把“样本”送入淋巴组织，启动相关免疫反应。但对于艾滋病病毒，由于神经节苷酯的存在，树突状细胞无法将其分割，因此病毒得以完整侵入免疫系统，这等于给免疫系统放置了一个允许艾滋病病毒自由出入的“特洛伊木马”。一旦进入淋巴，艾滋病病毒便将杀死人体的免疫细胞。
研究人员在新一期网络科学杂志《科学公共图书馆》上说，未来有望据此开发组织艾滋病病毒蔓延的新药物，配合常用的“鸡尾酒疗法”共同打击艾滋病病毒。不过由于需要进行完整的临床试验，新药物的研制和投放市场至少还需十几年。
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